Identification of serum metabolome signatures associated with retinal and renal complications of type 2 diabetes

Genetic Risk of Primary Aldosteronism and Its Contribution to Hypertension: A Cross-Ancestry Meta-Analysis of Genome-Wide Association Studies

Multi-trait and cross-population genome-wide association studies across autoimmune and allergic diseases identify shared and distinct genetic component

Differential and shared genetic effects on kidney function between diabetic and non-diabetic individuals

Multi-ancestry genetic study of type 2 diabetes highlights the power of diverse populations for discovery and translation

Genome-wide risk prediction of common diseases across ancestries in one million people

Response to Comment on Dawed et al. Genome-Wide Meta-analysis Identifies Genetic Variants Associated With Glycemic Response to Sulfonylureas. Diabetes Care 2021;44:2673-2682

The power of genetic diversity in genome-wide association studies of lipids

A deep learning method for HLA imputation and trans-ethnic MHC fine-mapping of type 1 diabetes

Cause-specific mortality rates in patients with diabetes according to comorbid macro- and microvascular complications: BioBank Japan Cohort

Subtype-specific gout susceptibility loci and enrichment of selection pressure on ABCG2 and ALDH2 identified by subtype genome-wide meta-analyses of clinically defined gout patients

Identification of type 2 diabetes loci in 433,540 East Asian individuals

Diabetes and cancer risk: A Mendelian randomization study

Identification of 28 new susceptibility loci for type 2 diabetes in the Japanese population

Protein-altering variants of PTPN2 in childhood-onset Type 1A diabetes

Genome-wide analyses identify a role for SLC17A4 and AADAT in thyroid hormone regulation

GWAS of clinically defined gout and subtypes identifies multiple susceptibility loci that include urate transporter genes

Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypes

Genome-wide association studies in the Japanese population identify seven novel loci for type 2 diabetes

A single nucleotide polymorphism in KCNQ1 is associated with susceptibility to diabetic nephropathy in japanese subjects with type 2 diabetes

A genome-wide association study in the Japanese population identifies susceptibility loci for type 2 diabetes at UBE2E2 and C2CD4A-C2CD4B

Replication study for the association of new meta-analysis-derived risk loci with susceptibility to type 2 diabetes in 6,244 Japanese individuals

SNPs in KCNQ1 are associated with susceptibility to type 2 diabetes in East Asian and European populations

Genetic drivers of heterogeneity in type 2 diabetes pathophysiology

Body mass index stratification optimizes polygenic prediction of type 2 diabetes in cross-biobank analyses

Characteristics of patients with liver cancer in the BioBank Japan project

Cholesterol levels of Japanese dyslipidaemic patients with various comorbidities: BioBank Japan

Serum glucose, cholesterol and blood pressure levels in Japanese type 1 and 2 diabetic patients: BioBank Japan

Statin use and all-cause and cancer mortality: BioBank Japan cohort

Construction of a population-specific HLA imputation reference panel and its application to Graves’ disease risk in Japanese

Survival of macrovascular disease, chronic kidney disease, chronic respiratory disease, cancer and smoking in patients with type 2 diabetes: BioBank Japan cohort

Genome-wide association study identifies three novel loci for type 2 diabetes

Genetic Risk, Healthy Lifestyle Adherence, and Risk of Developing Diabetes in the Japanese Population

The construction of risk prediction models using GWAS data and its application to a type 2 diabetes prospective cohort

A single-nucleotide polymorphism in ANK1 is associated with susceptibility to type 2 diabetes in Japanese populations

Association between single nucleotide polymorphisms within genes encoding sirtuin families and diabetic nephropathy in Japanese subjects with type 2 diabetes

Identification of independent risk loci for Graves’ disease within the MHC in the Japanese population